Thank you Kathy. I saw that too and saved it but had not had a chance to send it to you, but I did not know he was yet another CPS social worker!!!!! Just like the guy in NY who killed the UPS driver coming out of WalMart because he was sure it was Donald Trump! Like Donald Trump would ever shop at WalMart! Hello!!!! But he had just recently left a very high position with CPS!

Then with this case yesterday in the Facebook murder, the guy has a gambling addiction which is a huge clue that he is on antidepressants which cause the gambling so often! (Even the doctor from my small home town, who helped me to adopt my daughter, suffered a Prozac-induced gambling addiction before he killed his wife and himself and shocked all who knew him.)

This is what these poor foster kids have to deal with – all these super drugged up social workers who are losing it on their meds but everyone around them misses it because they are all drugged up as well! I have a whole long list of cases of these crazy social workers with stories that most would not believe they are so far out there and they have no oversight in their jobs where they have total say over all these poor children!

Kathryn A Gibb Kinley shared 3TV Phoenix’s live video.
28 mins
I saw this on Good Morning America this morning. In northern Ohio, he walked up to a perfect stranger, an elderly gentleman walking down the sidewalk, shot him in the face killing him, recorded it and posted it to Facebook. He also stated he has killed 13 others. Looks like he wants to commit suicide by cop. Also has stated he “lost everything” due to a gambling addiction, yet blamed his girlfriend who just broke up with him for driving him to murder. He was a counselor for a foster placement agency, I believe. Looks like we have another one….. this makes me so angry.
3TV Phoenix is live now.
2 hrs
WANTED: A nationwide manhunt is underway for the suspect accused of killing an elderly man in Cleveland and posting the murder on social media. Help get his face out everywhere so he can be captured. Developing information:





As you will see from these reports his explanation is far too similar to reports of someone experiencing antidepressant-induced homicidal ideation….



What so many were not aware of is that an increase in serotonin by an accompanying decrease in one’s ability to metabolize serotonin was long known to produce both impulsive murder and suicide. See this study out of the Southern California:

1996 – Mutant Mice May Hold Key To Human Violence – An Excess Of Serotonin.

08/11/1996 – Mutant Mice May Hold Key To Human Violence – An Excess Of Serotonin, A Chemical That Helps Regulate Mood And Mental Health, Causes Mayhem



What the world remains unaware of is the fact that 86% of those who are diagnosed with the most deadly sleep disorder known as REM Sleep Disorder (RBD) are currently taking antidepressants. REM Sleep Disorder is a condition in which there is no paralysis during sleep thus allowing the patient to act out the dreams or nightmares they are having. Tragically 80% of those going into this sleep disorder hurt themselves or others including both murder and suicide as a result.

This is possibly the most deadly of all reactions one can have to antidepressants. Even more frightening though is to learn that before the introduction of the SSRI antidepressants RBD was known mainly as a drug withdrawal effect. Thus the chances of going into this dangerous reaction should be expected to increase as one goes into withdrawal. This is why it is so important to avoid as much of the withdrawal effects as possible by tapering off the antidepressant very, very slowly. Feel free to join us on Facebook to learn more about this disorder on our Antidepressant-induced REM Sleep Disorder group:

Have a Joyous Easter Weekend!!!

I LOVE THIS SHORT LITTLE VIDEO WITH SUCH A PROFOUND MESSAGE!! Remember this is the opportunity His sacrifice offered to us all – to become our best and give it all we’ve got in love and service to all as He did by paying it forward!


Eat Like the Person You Want to Become

Eat Like the Person You Want to Become!Credit: EDEKA

Posted by Uplift Connect on Sunday, March 12, 2017

Antipsychotics Tied to Respiratory Failure in At-Risk Patients

Association Between Antipsychotic Agents and Risk of Acute Respiratory Failure in Patients With Chronic Obstructive Pulmonary Disease

Megan Brooks

January 12, 2017

Antipsychotics have been linked to respiratory failure in a dose-dependent manner among patients with chronic obstructive pulmonary disease (COPD), results of a large observational study show.

“Multiple cases about acute respiratory distress or acute respiratory failure [ARF] from the use of antipsychotics have been reported during the past decades. Nevertheless, no population-based studies have been conducted to examine this potential drug safety issue,” study investigator Meng-Ting Wang, PhD, of the National Defense Medical Center in Taipei, Taiwan, told Medscape Medical News.

“Clinicians should exercise caution when prescribing antipsychotics to patients with COPD and avoid high doses if possible,” the investigators write.

The study was published online January 4 in JAMA Psychiatry.

Dose-Dependent Relationship 

The researchers analyzed healthcare claims records in the Taiwan National Health Insurance Database and antipsychotic medication history for 5032 patients with COPD who developed incident and idiopathic ARF (excluding cardiogenic, traumatic, and septic causes).

Using a case-crossover study design, they compared antipsychotic use during the 2 weeks before the ARF event (case period) and an earlier control period 2 to 3 months before the ARF event.

A total of 590 (11.7%) patients with ARF filled at least one antipsychotic prescription during the case period, compared with 443 (8.8%) during the control period. This corresponds to a 1.66-fold (95% confidence interval [CI], 1.34 – 2.05; P < .001) adjusted increased risk for ARF, regardless of antipsychotic class and route of administration.

The risk for ARF with antipsychotic use was dose-dependent, increasing from a 1.52-fold risk for a low defined daily dose (DDD) of 0.25 or less to a 3.74-fold risk for a high DDD of 1 or more.

“Overall, this is the first population-based observational study that reports a dose-dependent acute effect of antipsychotic use on risk of ARF,” said Dr Wang.

The findings, he added, have important implications for the management of COPD patients.

“First, we urge healthcare professionals to be vigilant about the development of ARF in COPD patients receiving antipsychotic treatment, especially during the initial phase of treatment,” he said.

“Second, antipsychotic use in COPD patients needs to be justified, given we noticed a high proportion of off-label use in our population. Third, according to our dose analysis, high daily dose of antipsychotics with more than 1 DDD should be avoided, and the risk should not be overlooked even in patients at a dose as low as a quarter of 1 DDD,” Dr Wang added.

“Fourth, this novel finding of respiratory adverse events from antipsychotics should be considered when weighing benefits against risks of using antipsychotics in COPD patients, but patients are not suggested to discontinue antipsychotics without consulting their physicians. In addition, we advise COPD patients on antipsychotics not to neglect symptoms of breathing difficulty or respiratory abnormalities and should seek medical help as soon as possible,” he added.

Commenting on the findings for Medscape Medical News, Dr Janna Gordon-Elliott, psychiatrist at Weill Cornell Medicine and New York–Presbyterian Hospital in New York City, said the article is “notable because it highlights a risk with antipsychotics that I don’t think is high up on our radar. In reviewing the literature after reviewing this paper, I am struck by how little this risk is talked about,” said Dr Gordon-Elliott, who was not involved in the study.

“This correlational study doesn’t necessarily put the nail in the coffin and can’t prove causation, although they did a really nice job of controlling for a lot of factors, and with this large population sample, they do show that this is more than just a few case reports,” she added.

“What the paper doesn’t tell us is why these COPD patients were on antipsychotics, and that’s probably just beyond the scope of the paper. But antipsychotics are being used off label in a lot of patients, and this paper should really make us think twice about that in patients with lung disease,” Dr Gordon-Elliott said.

The study had no commercial funding. The authors have disclosed no relevant financial relationships.

JAMA Psychiatry. Published online January 4, 2017. Abstract


Today is supposedly World Bipolar Day. I propose that minus antidepressants we would never have seen a World Bipolar Day because NOTHING has caused more cases of Bipolar than antidepressants. Initially when the SSRI antidepressants were introduced many psychiatrists refused to prescribe Prozac or drugs like it due to their strong potential of inducing a manic psychosis. Prozac or drugs which work on the same principle as Prozac — Zoloft, Luvox, Paxil, Celexa etc., are known as selective serotonin reuptake inhibitors (SSRIs) and are designed to inhibit the metabolism of serotonin, thereby increasing levels of this neurotransmitter in the brain and throughout the body. Yet what so few are aware of is that the psychedelic drugs (PCP also known as Angel Dust and LSD) actually mimic serotonin in order to produce hallucinations. So as the level of serotonin increases in the system no one should be surprised to learn that they begin to suffer hallucinations and psychosis.

For example in the June 1999 edition of Clinical Psychiatry News, Dr. Malcolm Bowers, a psychiatrist at Yale had found that physicians are not paying enough attention to patient factors that could make treatment with SSRIs dangerous. He found that, “SSRI induced psychosis has accounted for eight percent of all hospital psychiatric admissions over a 14-month period…What is surprising is that this particular group of side effects is really underplayed.” That comes out to about a quarter of a million people in the late 90’s per year were being chemically induced into a Bipolar psychosis. He then said that the large majority were being undetected as antidepressant-induced psychosis and were mistakingly continued on the offending medications, meaning the drugs were being allowed to continue to exacerbate the Bipolar condition. I recall a case I worked on in the mid 90’s where a patient sued Eli Lilly for causing his Bipolar which cost him his entire inheritance. Dr. Fred Goodwin, so well known for his large text written on Bipolar Disorder testified in his behalf that this was Prozac-induced and he won his case against them.

But Dr. Bowers study was done close to two decades ago. But with the extremely widespread use of these drugs since that time it seems everyone now knows someone who has a diagnosis of Bipolar when before the introduction of the first SSRI, Prozac most of us had never heard of someone who was Bipolar or Manic-Depressive as it was once called.

Here I am discussing that and its impact upon society in the 2006 FDA hearing on antidepressants when the Black Box Warnings were increased to the age group of anyone under the age of 25:

The full transcript of this testimony before the FDA Advisory Committee in December of 2006 can be found here:

Bipolar is Actually a Sleep/Seizure Disorder

I also have a DVD on Bipolar explaining the evidence behind it being a Sleep/Seizure Disorder and detailing how antidepressants cause this Bipolar reaction to antidepressants. The cost is $4.95 and can be watched online. It also explains how there are natural ways to reverse this Bipolar reaction. That can be found here:

Facebook Group – Antidepressant-Induced Bipolar and Schizophrenia

We also have a Facebook group addressing this issue and acting as a support to those going through this reaction plus helping others to understand how easily this can happen especially during abrupt changes in dose of an antidepressant. That group can be found at this link:


The Picture Above is Frank After and This is Frank Before

How Frank Ferrante Went From Depressed, Suicidal, Seriously Ill

to Healthy in Only 42 Days!

So many of you were disappointed when I reposted the blog on the amazing transformation of Frank Ferrante in the movie “May I Be Frank?” because the link to the free viewing of the movie had expired. Well today I got an email from Food Matters who are offering a free viewing of the movie for the next 48 hours beginning this morning!!! So I am encouraging all of you to watch it to see the amazing changes one can make with diet and other alternatives in such a short time. Keep in mind that Frank not only had been on Effexor for 10 years, but during the same time period he also was being treated with Interferon for Hepatitis C, and I think he was also being medicated for diabetes. The Hep C was gone in 23 days! You will witness an incredible change first hand and Frank will keep you laughing through most of it.

(WORD OF CAUTION: If you are not accustomed to a NY Italian’s rough language, get ready to ignore it and watch the miracle of Frank becoming who he says is the person he came here to be!!) Our original post on Frank’s story is posted below.


Have you watched Part 5 of the workshop yet?

If not, click here to watch part 5 now! Then click the link in the upper right hand corner where it says “Watch Now”

There are such valuable insights during this part of the workshop, as we discuss more of the emotional side of cleansing and health journey. We can’t cleanse our bodies without cleansing our emotions, and both are incredibly valuable.

Make sure to check out my fun and inspiring chat with Frank Ferrante. Frank has a truly unique perspective on life and health and has come so far in his journey.

We want to thank you so much for joining us for this workshop, and also want to commend you for taking the time to educate and inspire yourself to live a healthier life.

So now you might be wondering, “How how do I put these learnings into action?” This can be the most difficult part of the path because we often don’t know what to do next!

Our 7 Day Food Matters Cleanse is the perfect next step for you, if you are ready to start rebalancing your body and removing toxins that are holding you back from experiencing ultimate health and vitality.

It’s so easy to let our health be put on the “backburner” and not realise that we are robbing ourselves and those we love of our best self. I often think back and wonder where my father would be today, if he had not taken action and made his health his #1 priority! It’s kinda scary to think about…



U.S. Suicide Rate Surges to a 30-Year High

“It’s really stunning to see such a large increase in suicide rates affecting virtually every age group,” said Katherine Hempstead, senior adviser for health care at the Robert Wood Johnson Foundation

If these statistics say nothing else, they at least make it clear that antidepressants do not work. And apparently the only ones surprised by this are the so called “experts” in suicide who have vested interests in Pharma and are therefore, turning a blind eye to statistics that have been starring them in the face for decades. Anyone familiar with my work on the dangers of antidepressants have expected this for some time now &amp; witnessed it all around them. I encourage you to read this to see how bad it has become and before doing so I am going to share with you the testimonies of two of my good friends who testified to the FDA in 1991 that this was happening as a direct result of Prozac – the mother drug of this whole new generation of SSRI and SNRI antidepressants. We now have dozens of prescription drugs on the market which have warnings that they cause suicide. We have antidepressants, atypical antipsychotics, acne medications, asthma medications…the list is so very long! Even Tamiflu was removed from the market in Japan due to the suicides from that drug.

Anyone familiar with my work on the dangers of antidepressants have expected this for some time now & witnessed it all around them. I encourage you to read this to see how bad it has become. but before doing so I want you to see the video testimonies of two of my good friends testifying before the FDA in 1991. They warned that these suicides were increasing drastically as a direct result of Prozac – the mother drug of this whole new generation of SSRI & SNRI antidepressants & atypical antipsychotic drugs.

We now have dozens of prescription drugs on the market which have warnings that they cause suicide. We have antidepressants, atypical antipsychotics, acne medications, asthma medications…the list is so very long! Even Tamiflu was removed from the market in Japan due to the suicides from that drug. And about everyone in this country has access to a drug pusher they see regularly who they refer to as their personal physician while nurses share that 75% of the physicians and nurses they work with take antidepressants.

The first in sharing her experience almost three decades ago, in an attempt to prevent this nightmare, is LeAnne Westover, wife of singer Del Shannon (Charles Westover)…please click the following link to watch her testimony…

1991 FDA Antidepressant Hearing: Le Anne Westover Testifies of Husband Del Shannon’s Prozac Suicide

Second is Maria Malakoff, a Florida pharmacist, testifying about both her own suicide attempt on Prozac and the suicide of her husband, also a pharmacist. Maria warned the FDA back in 1991 that the day would come that every family in America would be affected by antidepressants if they did not issue warnings back then. Tragically her statement has proved prophetic!

Their pharmacy was the first pharmacy to remove Prozac from their shelves…

1991 FDA Antidepressant Hearing: Suicide & Suicide Attempt by Pharmacist Maria Malakoff

U.S. Suicide Rate Surges to a 30-Year High

“It’s really stunning to see such a large increase in suicide rates affecting virtually every age group,” said Katherine Hempstead, senior adviser for health care at the Robert Wood Johnson Foundation

April 22, 2016

WASHINGTON — Suicide in the United States has surged to the highest levels in nearly 30 years, a federal data analysis has found, with increases in every age group except older adults. The rise was particularly steep for women. It was also substantial among middle-aged Americans, sending a signal of deep anguish from a group whose suicide rates had been stable or falling since the 1950s.

The suicide rate for middle-aged women, ages 45 to 64, jumped by 63 percent over the period of the study, while it rose by 43 percent for men in that age range, the sharpest increase for males of any age. The overall suicide rate rose by 24 percent from 1999 to 2014, according to the National Center for Health Statistics, which released the study on Friday.

The increases were so widespread that they lifted the nation’s suicide rate to 13 per 100,000 people, the highest since 1986. The rate rose by 2 percent a year starting in 2006, double the annual rise in the earlier period of the study. In all, 42,773 people died from suicide in 2014, compared with 29,199 in 1999.

Graphic | A Growing, Widespread Toll BY THE NEW YORK TIMES

“It’s really stunning to see such a large increase in suicide rates affecting virtually every age group,” said Katherine Hempstead, senior adviser for health care at the Robert Wood Johnson Foundation, who has identified a link between suicides in middle age and rising rates of distress about jobs and personal finances.

Researchers also found an alarming increase among girls 10 to 14, whose suicide rate, while still very low, had tripled. The number of girls who killed themselves rose to 150 in 2014 from 50 in 1999. “This one certainly jumped out,” said Sally Curtin, a statistician at the center and an author of the report.

What to Do If You Need Help

American Indians had the sharpest rise of all racial and ethnic groups, with rates rising by 89 percent for women and 38 percent for men. White middle-aged women had an increase of 80 percent.

The rate declined for just one racial group: black men. And it declined for only one age group: men and women over 75.

The data analysis provided fresh evidence of suffering among white Americans. Recent research has highlighted the plight of less educated whites, showing surges in deaths from drug overdoses, suicides,liver disease and alcohol poisoning, particularly among those with a high school education or less. The new report did not break down suicide rates by education, but researchers who reviewed the analysis said the patterns in age and race were consistent with that recent research and painted a picture of desperation for many in American society.

“This is part of the larger emerging pattern of evidence of the links between poverty, hopelessness and health,” said Robert D. Putnam, a professor of public policy at Harvard and the author of “Our Kids,” an investigation of new class divisions in America.

The rise in suicide rates has happened slowly over many years. Federal health researchers said they chose 1999 as the start of the period they studied because it was a low point in the national suicide rate and they wanted to cover the full period of its recent sustained rise.

The federal health agency’s last major report on suicide, released in 2013, noted a sharp increase in suicide among 35- to 64-year-olds. But the rates have risen even more since then — up by 7 percent for the entire population since 2010, the end of the last study period — and federal researchers said they issued the new report to draw attention to the issue.

Policy makers say efforts to prevent suicide across the country are spotty. While some hospitals and health systems screen for suicidal thinking and operate good treatment programs, many do not.

“We have more and more effective treatments, but we have to figure out how to bake them into health care systems so they are used more automatically,” said Dr. Jane Pearson, chairwoman of the National Institute of Mental Health’s Suicide Research Consortium, which oversees the National Institutes of Health funding for suicide prevention research. “We’ve got bits and pieces, but we haven’t really put them all together yet.”

She noted that while N.I.H. funding for suicide prevention projects had been relatively flat — rising to $25 million in 2016 from $22 million in 2012 — it was a small fraction of funding for research of mental illnesses, including mood disorders like depression.

The new federal analysis noted that the methods of suicide were changing. About one in four suicides in 2014 involved suffocation, which includes hanging and strangulation, compared with fewer than one in five in 1999. Suffocation deaths are harder to prevent because nearly anyone has access to the means, Ms. Hempstead said. And while the share of suicides involving guns declined — guns went from being involved in 37 percent of female suicides to 31 percent, and from 62 percent to 55 percent for men — the total number of gun suicides increased..

The question of what has driven the increases is unresolved, leaving experts to muse on the reasons.

Julie Phillips, a professor of sociology at Rutgers who has studied suicide among middle-aged Americans, said social changes could be raising the risks. Marriage rates have declined, particularly among less educated Americans, while divorce rates have risen, leading to increased social isolation, she said. She calculated that in 2005, unmarried middle-aged men were 3.5 times more likely than married men to die from suicide, and their female counterparts were as much as 2.8 times more likely to kill themselves. The divorce rate has doubled for middle-aged and older adults since the 1990s, she said.

Disappointed expectations of social and economic well-being among less educated white men from the baby-boom generation may also be playing a role, she said. They grew up in an era that valued “masculinity and self-reliance” — characteristics that could get in the way of asking for help.

“It appears this group isn’t seeking help but rather turning to self-destructive means of dealing with their despair,” Professor Phillips said.

Another possible explanation: an economy that has eaten away at the prospects of families on the lower rungs of the income ladder.

Dr. Alex Crosby, an epidemiologist at the Centers for Disease Control and Prevention, said he had studied the association between economic downturns and suicide going back to the 1920s and found that suicide was highest when the economy was weak. One of the highest rates in the country’s modern history, he said, was in 1932, during the Great Depression, when the rate was 22.1 per 100,000, about 70 percent higher than in 2014.

“There was a consistent pattern,” he said, which held for all ages between 25 and 64. “When the economy got worse, suicides went up, and when it got better, they went down.”

But other experts pointed out that the unemployment rate had been declining in the latter period of the study, and questioned how important the economy was to suicide.

The gap in suicide rates for men and women has narrowed because women’s rates are increasing faster than men’s. But men still kill themselves at a rate 3.6 times that of women. Though suicide rates for older adults fell over the period of the study, men over 75 still have the highest suicide rate of any age group — 38.8 per 100,000 in 2014, compared with just four per 100,000 for their female counterparts.

Vitamin C Miracle Natural Cure for Pneumonia, Cancer & Adrenal Fatigue/Failure & ???

Living Proof – “60 Minutes” New Zealand – The Allan Smith Story – TV Documentary

(Click the second picture to view, not this one)

Allan Smith, a New Zealand Dairy farmer, contracted Swine Flu while away on vacation in Fiji.  When he returned home, the flu quickly evolved into severe pneumonia which left him in a coma on Life Support in the Intensive Care Unit.  Chest Xrays showed the lungs were completely filled with fluid with an “opaque” appearance called “white out”.  After three weeks of this, Allan’s doctors asked the family permission to turn off the machines and let him die.  Allan’s wife Sonia had a brother with some medical knowledge, so he stepped in and said, “you haven’t tried everything, You have got to try high dose IV vitamin C on Allan”.  At first, the doctors resisted, saying it was useless. Next, the three sons weighed in with a persuasive argument to try the IV vitamin C, saying there was nothing to lose.

Vitamin C Saves Man Dying of Viral Pneumonia by Jeffrey Dach MD…. [NOTE: Although this report does not go into detail about the type of Vitamin C used it should be pointed out that far too many brands are made with GMO corn which explains why I do not work for me at all. The one I use is organic – big difference! The other does absolutely nothing for me. The difference in the two is more than obvious for me.]

Who Needs Antibiotics When Vit C Reverses Pneumonia in Three Hours?

Dr. Andrew Saul, an internationally-recognized expert in nutritional therapies and Jonathan Landsman talking about a simple, yet powerful way to cure pneumonia in 3 hours – naturally.

Dr. Saul reveals (2) incredible stories of recovery – from serious health problems – without the use of toxic medications or risky surgical procedures. One story is about how to reverse viral pneumonia …

and the other illustrates the healing power of nutritional supplements for babies. After listening to this video – you’ll never look @ the seasonal flu shot the same – again.

To hear the rest of this interview and gain INSTANT access to over 300 shows with over 200 integrative healthcare providers – join us inside the NaturalHealth365 INNER CIRCLE.

To join – simply click the link below:

Vitamin C Helps Adrenal Function Which Is Often Impaired By Antidepressant Use

Another of our many Facebook groups addresses an all too common after effect of antidepressant use which Vitamin C can help … “Antidepressant-Induced Adrenal Fatigue/Failure” which can be found at the following link:

One of the most early studies done on Prozac, the mother drug of all the SSRI and SNRI antidepressants, was a study indicating that only one single dose of 30mgs of Prozac DOUBLED the levels of cortisol, often referred to as the “death hormone.” The deliterious effects of increasing cortisol are extensive including the fact that elevated cortisol is the major marker indicating depression!

Vitamin C (Ascorbic Acid) is another crucial vitamin in adrenal function and maintenance of healthy levels of cortisol and DHEA is vitamin C (Bornstein 2004; Morfin 2002).

Deficiencies of this vitamin can have profound effects on adrenal function (Brody 2002; Carroll 2000). The benefits of vitamin C are multiple, acting as an anti-inflammatory and co-factor in soft tissue synthesis and repair (Eipper 1992; Hemila 1996; Evans 2008).

In addition, ultra-marathon runners who were given 1,500 mg vitamin C after a race displayed less dramatic elevations in cortisol and epinephrine levels than is typical after such extreme stress (Peters 2001). Moreover, this same study found that vitamin C was able to suppress inflammation in the runners as well.

MEDS: Yet Another Son Decapitates Mother, Greets Police With Head in One Hand, Knife in the Other

Oliver Funes-Machado, made a 911 call Monday afternoon to report he had killed his mother because “he felt like it”. (Do you remember the definition of “homicidal ideation” I discussed in my last post? You might want to review that.) Four years ago young Oliver began having shyness issues. Only days before this tragedy was he released from a psychiatric facility where he had been by a court order.

He was taken into custody without resistance after putting down his 35-year-old mother’s head. The rest of her body was found inside the home.

Machado’s lawyer said the Honduran native is “profoundly disturbed.” Obviously that is an understatement.

Search warrants show that deputies found four medications labeled as being for the suspect on a bedroom dresser. And according to court documents, Machado took four different medications for psychosis and schizophrenia.

I will take bets that what happened in that psych facility he was just released from his meds we’re changed also. There was an increase or decrease in dose of one or more of those meds, or a new one or several new ones were added to what he was taking – just as both the Effexor & Remeron Andrea Yates was taking were drastically changed in dosage just the day before she drowned all five of her children. 

(These drastic changrs in dose took her to the maximum doseof each drug: Effexor was decreased by 1/3 down from 450mg – 1 1/2 times the maximum dose to the maximum dose of 300mg while Remeron was increased by 1/4 from 45mg to the maximum dose for that drug of 60mg.)  

And just as Brynn Hartman’s doctor cut her Zoloft in half just three weeks before she shot her husband & herself. And another young man, Derek Ward, abruptly discontinued his medication four days earlier while waiting to get into his doctor for a new antipsychotic medication when he decapitated his mother – a popular college professor. He then left her body lying in the street while he kicked her head down the street, and walked a block to the subway where he jumped in front of a train killing himself. Although far from the only cases like this Derek’s is the case I thought of when I saw this one. (See more on Derek’s case below…and be sure to read the reaction of a close family friend who understands the extreme dangers of these serotonergic drugs.)          

Miriam Banegas, a longtime friend of the Funes-Machado family, said she is heartbroken over the loss of Yesenia Funes Beatriz Machada and concerned for the son, Oliver, who is accused in the killing.

“He did it with his hands, but he didn’t do it with his heart. Like it was a product of something else,” Banegas said Wednesday through a translator.

As Kids Cry in Background, North Carolina Teen Accused of Decapitating Mom Confesses to Killing Her in Chilling 911 Call (Warning: Graphic Content)…gally/1789038/

 Read the following comment from a friend of the family added to our post on this case about a year and a half after it happened…

Posted on November 6, 2014

Derek Ward & his mother, Pat Ward

UPDATE ON THIS CASE: A personal letter to me from a family friend who knew this boy and his mother for years….”People do not understand what is happening as a result of these drugs. They do not question murders and suicide. They just chalk it up to “oh crazy people” and go on with their lives. This upset me greatly.

“In the spring of 2014 I heard on the news: Farmingdale man murders woman – decapitates her head – kicks it down the street, then walks a block away and throws himself in front of a train. Suicide? In my town? Crazy people, I think…

But as it turned out…“This “man” was my son’s childhood friend and a neighbor. I coached his mother through natural childbirth and breastfeeding. He was a very much wanted child. Derek Ward was in my house every day with my son playing outside in their childhood days. This incident comes too close to home. I was shocked and traumatized by this. So is my son.”


HOW SAD!!! He looked like such a sweet kid in this picture obviously taken before the 10 years of antidepressants! Isn’t it amazing these drugs are such monster making meds! It sounds like he was medicated most of his life! His uncle said he had dyslexia and ADD which generally means you are given Ritalin or something similar – stimulant drugs known to cause depression.

Then it appears he was treated for 10 years for depression and apparently went psychotic on those meds so the mother was trying to get him in for a new medication for schizophrenia – the appointment two days away when he killed her and himself.

He was off his meds for four days!!! Does that give anyone any idea why our website has warned since 1995 that coming off one of these drugs can be more dangerous than staying on them unless you know how slowly to wean off and what you are doing? Going on, coming off, or switching your antidepressant is the most dangerous time of taking the drugs! The FDA has warned that any abrupt change in dose of an antidepressant, whether increasing or decreasing the dose, or switching medications where you are both decreasing one and increasing another, can cause suicide, hostility, or psychosis – all of which appeared in this case! And once again I point out that it took only only four days off medication for this deadly reaction!

So why is this warning something doctors seem to be oblivious to and do not warn their patients about?! How many cases like this do I have to document before the world wakes up to this antidepressant-induced nightmare?! Here is just a sampling of the documented cases so far: www.ssristories.NET.

The data on this horrific reaction is so clear that drug company experts are compelled in court to testify that these drugs which impair the metabolism of serotonin (serotonin reuptake) do produce impulsive murder and suicide. (See the mass murder/suicide case of the Donald Schell family in the WY wrongful death case of Tobin vs Glaxo)

And read below a 1996 study discussed in the LA TIMES showing how these drugs, designed to increase levels of serotonin produce such incredible violence in those never before known to demonstrate such violence…

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In 2004 the FDA held two hearings on antidepressants and suicide, mainly in children. One was February 2, 2004 and the other September 13, 2004. They concluded that a Black Box Warning should be on the label of all antidepressants – any that had ever been approved and any to be approved in the future. We are gradually posting all of those testimonies on our website at for the world to hear what the FDA heard that day from those who suffered it and from loved ones who lost loved ones to this deadly side effect. They also warned that abrupt changes in dose of an antidepressant, whether an increase or decrease, could cause suicide, hostility, or psychosis. And within a very short time added the suicidal ideation warning to many other medications including the serotonergic atypical antipsychotics and the anti-seizure medications including drugs like Neurontin (Gabapentin) which seems to be being prescribed for anything you can think of in spite of Pfizer paying out unheard of fines for encouraging doctors to prescribe off label for things it has never been approved for. They have even been found guilty criminally for racketeering (RICO) over this.

Sadly since 2004 this warning about suicidal ideation has been downplayed by Pharma and doctors on a regular basis to patients and their loved ones. Few even know what suicidal ideation or homicidal ideation is. So, we have started a new Facebook group for both side effects at: 

Suicidal and homicidal ideations are not just the act of killing oneself or others. It is a continuous compulsion with ruminating thoughts of both killing and ruminating thoughts of various methods of doing so. So ruminating and continuous that one cannot think of much of anything else. Here are two real life experiences from patients in their own words to give you an example of both antidepressant-induced suicidal ideation & antidepressant-induced homicidal ideation-reports I have listened to for over two decades from people age 5 to 90:

“Homicidal Ideation” Described by Patient Who Suffered

This Antidepressant-Induced Side Effect

“Only two days on Zoloft and I wanted death bad – I wanted death like I wanted a new Ferrari…

but I wanted it not just for me, but for everyone!!!”

“I am a veteran of 15 years. I have seen the disasters from prescription medications especially the ones we were issued to treat PTSD and other mental illnesses. A couple of years ago, I agreed to take Zoloft as suggested by my doctor for depression. I had denied it for years.

“Within 2 days I waas truly suicidal. I wanted death bad. It wasn’t like the normal suicidal thoughts where I felt so depressed I would rather be dead, it was more like I wanted death like I wanted a new Ferrari. But not just for me, I wanted it for everyone! I wanted to get my friends together and thought let’s all die together. I have been confused by how this drug can make you feel this way. I certainly felt why so many commit suicide on these drugs and why it is a listed side effect! I tried to OD, but I sucked at it fortunately. I never touched another pill again.

“I got out of the Army last year and have been on a spiritual journey for myself.” … C. C. D.


“Suicidal Ideation” Described by Patient Who Suffered

This Antidepressant-Induced Side Effect

” Suicidal compulsions from these drugs are strong, overwhelming and almost impossible to fight!”

“I can tell you from experience because it happened to me. I was never suicidal. I never had anxiety. I took these drugs for over 20 years but then it happened. The drug affected my mind and my brain and my central nervous system. When the suicidal thoughts are caused from the medicine they are strong and overwhelming and almost impossible to fight. If people could feel just for one hour how you feel when this happens then they would understand and all these drugs would be pulled from the market immediately. It’s not the same as being sad and feeling like you don’t want to go on. It’s more like something telling you how worthless and no good you are and you have to die over and over. It affects your emotions and all your thoughts. Think the worst saddest most horrible thing that ever happened to you then having your brain stuck in that place and be unable to feel any positive emotion. That’s how it feels.

“I hated guns and would never even think about owning one but the thought that was in my head over and over and over was to get a gun and kill myself. That’s not even like me. There were times that it would not let up. I would scream out loud “Stop! Leave me alone” still the thoughts persisted. That’s why I was so afraid to be alone. Your mind is completely changed ( physically changed) so that you can see/feel nothing good. Your whole reality is changed . Your thoughts are all that seems real when it happens. Nothing else.

“The worst part is you feel like you are worthless and shouldn’t be alive. Your mind is so confused that you don’t even realize that it’s the medicine. I STRONGLY feel that there should be more warnings besides just saying it can make you suicidal. My idea of suicidal ideation was much different than what happened to me. I didn’t realize that it would be an compulsion to die that ruminates over and over in your head and allowing few other thoughts to enter . That is what should be listed in the warning.”… Helen Marie Daff


Elevated Serotonin Has Long Been Linked To Violence

1996 Study – Mutant Mice May Hold Key To Human Violence

– An Excess Of Serotonin.

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Of Mice and Mayhem

The Fierce Tempers of Mutant Rodents Born With Their Brains Awash in the Chemical Serotonin May Provide a Clue to Violent Behavior Among Humans


The scientist grabs Mutant #9 by the tail, lifts the mouse out of its shoebox-size cage, and lowers it gently into another, identical container, the reeking, sawdust-floored home of Mutant #4.
Blind and jittery, the mice are freaks of nature, products of a genetic engineering experiment that did not go exactly as planned. But, oddly, their encounter in this fifth-floor laboratory at the USC School of Pharmacy may reveal something vital about human nature.

They square off, sniffing furiously, then inch closer. Within seconds, #9 corners #4. And then they dive at each another–a rolling, squeaking, clawing gray blur. Sawdust and fur fly.

Jean Chen Shih, a USC biochemist and promoter of this unlikely murine bout, jumps back, startled by the attack even though she was expecting it. “Normal mice fight also, but not so rapidly as these,” Shih says.

By any measure, the mice, called Tg8, are among the most aggressive in captivity.

This odd little spectacle is part of the quest for answers to the violence clawing at American’s soul. A Tg8 is born with its brain awash in an excess of serotonin, a neurotransmitter chemical that helps regulate mood and mental health, and Shih and her co-workers believe that that excess greatly contributes to the mouse’s fierce temper.

To be sure, a brawl between blind mice in an ivory tower is a far cry from the mayhem and brutality perfected by such brainy animals as ourselves. But the work does appear to touch on human experience: The Tg8’s cardinal biochemical defect was originally discovered in numerous related Dutchmen who committed arson, attempted rape and assault.

The Tg8 mice are the first laboratory animals to share both the biochemical defect and the behavior observed in a pedigree of violent criminals. In that sense, the mice are an important new tool for probing the physiology of running amok. By studying the mice’s trigger-happy biology, researchers hope to understand aggression better and perhaps develop new drugs that control it.

But for every potential new use of such information, critics envision a new abuse. Steven Rose, a biologist at the Open University in England, is an outspoken critic of the idea that one’s genetic makeup determines behavior–a scientific premise he calls “neurogenetic determinism.”

Sociologist Dorothy Nelkin of New York University says that conservatives might seize on biological explanations of violence to “dismantle the welfare state,” because controlling aggression with drugs could well be much cheaper than rehabilitation programs.

She also fears that if certain biochemical signatures became associated with violence or criminal behavior, people with such a makeup could be wrongly implicated and stigmatized as potential threats to society–the physiological equivalent of a bad credit rating. The Tg8 research and similar studies, she says, “open up a whole set of problems that are worthy of careful consideration.”

No such heady dilemmas weighed upon scientists at the Pasteur Institute near Paris when they accidentally created the Tg8 mouse strain two years ago. Olivier Cases and colleagues were trying to develop a novel gene therapy by injecting a one-celled embryo of a special lab strain of blind mice with a shred of foreign DNA. But instead of resulting in a “new” mouse pup with a bolstered immune system, the experiment led to a strain of male mice with a really bad temper.

The first indicator of that ill nature was painfully obvious: The mice nipped the researchers’ fingers. When caged together, male Tg8s–the Tg is for “transgenic”–tore each other apart. And the researchers also found that when male and female mice mated, the males were especially quick, grabby and forceful, eliciting more female squeaks, on average, than other males did.

Those traits may be reminiscent of any number of men, but the French researchers were put in mind of certain Dutch males in one extended family described in the medical literature. Over four generations, a remarkable number of those males were accused or convicted of rape, assault and arson, leading local psychologists as well as law enforcement authorities to watch them very closely.

After much study, Dutch scientists reported a finding in 1993 that, they believed, helped explain the aggressive males’ behavior: They were missing an enzyme called monoamine oxidase A, or MAO-A, which breaks down a variety of neurotransmitters, including serotonin. Lacking the MAO-A enzyme, the affected males, who also had borderline mental retardation, had extra-high levels of several neurotransmitters, including serotonin.

The genetic defect found in the violent Dutchmen is probably very rare, researchers say, and certainly doesn’t account for what makes most aggressive people act that way. Still, the finding offers a unique window into how disruptions in brain chemistry can be correlated with a pattern of antisocial behavior.

Given the Dutch findings, then, it was logical for the sore-fingered French researchers to want to know if their mutant mice also lacked the MAO enzyme. That’s where Shih, a world expert on that family of enzymes, came in. Not long after she agreed to test the Tg8 mice for the enzyme, a shipment of the creatures arrived from France (having spent weeks in quarantine at Los Angeles International Airport).

It took Shih and her co-workers several months of painstaking lab work to establish that the male mice were indeed lacking the gene for the MAO-A enzyme–just like the affected Dutchmen. “When this gene is missing, the animals are very aggressive and hyperactive,” Shih says.

Her Tg8 study, says Randy Nelson, a behavioral psychologist at Johns Hopkins University, was “one of the first to show a biological mechanism for aggressive behavior in an animal.” Follow-up studies published this spring in the journal Neuron suggest that the neurotransmitter defect actually affects the structure of the Tg8’s brain, most likely by skewing growth and development in fetal and newborn mice.

Nelson says it’s no surprise that genes affect temperament. “Anybody who knows the difference between a pit bull and a Labrador retriever knows that aggressive behavior has a genetic basis,” he says.

Some researchers expect that the biochemical analysis of behavior will pay off. Dr. Frederick Moeller, a psychiatrist at the University of Texas in Houston, hopes that the missing-enzyme research leads to new drugs for treating criminals and other violent people who simply can’t control their aggression. “I treat individuals who . . . can’t keep from assaulting hospital staff even long enough to get out of the hospital,” he says.

“The goal isn’t to control everybody and make them less aggressive,” he says. “The goal is to work with real people who have a real problem with aggression.”

For her part, Shih is a little dismayed to find herself in the middle of such a hot controversy. “I like to avoid the political issues,” the biochemist says.

Even though she is no sociologist, Shih believes that she can make a contribution to understanding human behavior by studying the Tg8’s biology.

That possibility is apparent to her whenever she returns the mice to their cages in the locked, windowless animal room across the hall from her office. Normal mice fare perfectly well living four to a cage. But the Tg8 males are held in solitary confinement, too hostile for mouse society.


08/11/1996 • Mutant Mice May Hold Key To Human Violence–An Excess Of Serotonin, A Chemical That Helps Regulate Mood And Mental Health, Causes Mayhem

Jean Chen Shih

Portland Press Herald

A Tg8 is born with its brain awash in an excess of serotonin, a neurotransmitter chemical that helps regulate mood and mental health, and [Jean Chen] Shih and her co-workers believe that that excess greatly contributes to the mouse’s fierce temper.

Mutant Mice May Hold Key To Human Violence–An Excess Of Serotonin, A Chemical That Helps Regulate Mood And Mental Health, Causes Mayhem

Jean Chen Shih

Portland Press Herald

A Tg8 is born with its brain awash in an excess of serotonin, a neurotransmitter chemical that helps regulate mood and mental health, and [Jean Chen] Shih and her co-workers believe that that excess greatly contributes to the mouse’s fierce temper.

The scientist grabs Mutant 9 by the tail, lifts the mouse out of its cage, and lowers it into another, identical container, the reeking, sawdust-floored home of Mutant 4. Blind and jittery, the mice are freaks of nature, products of a genetic engineering experiment that did not go exactly as planned. But, oddly, their encounter in this fifth-floor laboratory at the University of Southern California School of Pharmacy may reveal something vital about human nature. This odd little spectacle is part of the quest for answers to the violence clawing at America’s soul. A Tg8 is born with its brain awash in an excess of serotonin, a neurotransmitter chemical that helps regulate mood and mental health, and [Jean Chen] Shih and her co-workers believe that that excess greatly contributes to the mouse’s fierce temper.

SSRI RISK GREATER THAN BENEFIT: New Systematic Review of Research – SSRIs vs Placebo

Selective serotonin reuptake inhibitors versus placebo in patients with major depressive disorder. A systematic review with meta-analysis and Trial Sequential Analysis

Published February 8, 2017:

” We have clearly shown that SSRI significantly increase the risks of both serious and several non-serious adverse events. The observed harmful effects seem to outweigh the potential small beneficial clinical effects of SSRI, if they exist.


SSRIs versus placebo seem to have statistically significant effects on depressive symptoms, but the clinical significance of these effects seems questionable and all trials were at high risk of bias. Furthermore, SSRIs versus placebo significantly increase the risk of both serious and non-serious adverse events. Our results show that the harmful effects of SSRIs versus placebo for major depressive disorder seem to outweigh any potentially small beneficial effects.

Brief Overview of the Study

Highlights of the study bolded and with added emphasis:


The evidence on selective serotonin reuptake inhibitors (SSRIs) for major depressive disorder is unclear.


Our objective was to conduct a systematic review assessing the effects of SSRIs versus placebo, ‘active’ placebo, or no intervention in adult participants with major depressive disorder. We searched for eligible randomised clinical trials in The Cochrane Library’s CENTRAL, PubMed, EMBASE, PsycLIT, PsycINFO, Science Citation Index Expanded, clinical trial registers of Europe and USA, websites of pharmaceutical companies, the U.S. Food and Drug Administration (FDA), and the European Medicines Agency until January 2016. All data were extracted by at least two independent investigators. We used Cochrane systematic review methodology, Trial Sequential Analysis, and calculation of Bayes factor. An eight-step procedure was followed to assess if thresholds for statistical and clinical significance were crossed. Primary outcomes were reduction of depressive symptoms, remission, and adverse events. Secondary outcomes were suicides, suicide attempts, suicide ideation, and quality of life.


A total of 131 randomised placebo-controlled trials enrolling a total of 27,422 participants were included. None of the trials used ‘active’ placebo or no intervention as control intervention. All trials had high risk of bias. … SSRIs significantly increased the risks of serious adverse events…. This corresponds to 31/1000 SSRI participants will experience a serious adverse event compared with 22/1000 control participants. SSRIs also significantly increased the number of non-serious adverse events. There were almost no data on suicidal behaviour, quality of life, and long-term effects.


SSRIs might have statistically significant effects on depressive symptoms, but all trials were at high risk of bias and the clinical significance seems questionable. SSRIs significantly increase the risk of both serious and non-serious adverse events. The potential small beneficial effects seem to be outweighed by harmful effects.

Link to full study:


As Patents Expire Truth Begins to Surface

As I pointed out first in the 1994 issue of my text on antidepressants, “Prozac: Panacea or Pandora?,” the truth about the adverse effects of medications generally do not surface until the expiration of patents on these drugs – the time that the income on these drugs drastically drops as the majority of drugs in a group of drugs begins to go generic. Are we finally beginning to see that after years of warning of these deadly side effects? If so it is time to really begin warning about the deadly effects of withdrawal done too rapidly. We could be facing a nightmare of people wanting to withdraw from these drugs when most doctors know so very little about safe withdrawal from antidepressants.

PLEASE let people know to wean extremely gradually down off these drugs! For two decades the warning above has been at the top of our website warning of abrupt or rapid withdrawal. The body and brain need time to readjust after so many chemical changes!

Keep in mind the case of Traci Johnson, the young healthy volunteer (no depression), who took the antidepressant Cymbalta for 20 days and then after a withdrawal period of only four days hung herself in the laboratory for Eli Lilly, the manufacturer. After that tragedy and subsequent wrongful death suit Eli Lilly changed their time period for withdrawal in their clinical trials by double! So after 20 days on they began taking 8 days to wean off – almost the exact amount of time we have found over the years is a safe period of time to withdraw without serious reactions is about half the amount of time on the drug.